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Current evidence suggests that inflammation contributes to EDC through upregulation of central inflammatory cytokines, overactivation of microglia and astrocytes, and hyperactivation of the HPA axis, leading to neurotransmitter imbalance, impaired synaptic plasticity, increased pyroptosis, and OSpositioning EDC as a prototypical inflammation-driven and stress-driven neuropsychiatric comorbidity (Xu et al., 2018
Not enough blood reaching the brain could ultimately result in a stroke
Oxidative stress in the pathogenesis of skin disease
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Wie wirkt Glutathion auf das Immunsystem
245 MT predominantly resides in the cytoplasm
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