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We therefore propose that tissue-associated oxidative stress may promote conversion of circulating TTR into dsNNTTR locally within the interstitial compartment, rather than exclusively during intracellular synthesis or processing

Metformin suppressed oxidative stress through promoting Nrf2, NQO1, and HO-1 expression in MIA-induced mouse model Oxidative stress is a major causative factor in OA development 30

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