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Overexpressing CaBPR1 in tobacco conferred increased tolerance to the oomycete pathogen Phytophthora nicotianae , and the bacterial pathogens Ralstonia solanacearum and P

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PAR4 antagonist ML354 inhibited OGD-induced increase in Ca 2+ activity and decreased Bax/Bcl-2 ratio, reducing neuronal excitotoxicity [197,198,199]

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