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Because of this precision, they tend to have: High specificity for their targets Shorter halflives (they are broken down relatively quickly) Typically lower immunogenicity (less likely to provoke immune reactions) than larger biologic drugs

The real battle is won through effective

IGF-1 LR3 retains the ability to bind IGF-1 and insulin receptors but has reduced binding affinity to insulin-like growth factor-binding proteins (IGFBPs)

The emergence of Anifrolumab underscores the potential for targeting type-I IFN in the treatment of SLE