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However, inhibiting ferroptosis in hepatocytes can alleviate liver damage, suggesting that ferroptosis might represent a potential therapeutic target for HBV-ALF

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Notably, our lead compound KL1 reduced ROS/RNS production in macrophages, sensitized intracellular persisters to antibiotics, and enhanced antibiotic efficacy in murine infection models

Cagrilintide signaling triggers neurochemical responses that promote long-term satiation curves, allowing researchers to study central appetite regulation independently from standard glucagon-like peptide-1 (GLP-1) pathways