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This metabolic state arises from a dysregulation between uric acid production and elimination, culminating in its accumulation in the bloodstream

No significant cross-reactivity or interference between Glutathione (GSH) and analogues was observed

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Identification of tissue transglutaminase as the autoantigen of celiac disease

Second, multimodal therapies combining urate-lowering agents, anti-inflammatory cytokines, and immunomodulators within a single platform may synergize to halt disease progression while minimizing systemic side effects

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