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10.1016/J.YEXMP.2018.09.003 203 ZamiriK.KesariS.PaulK.HwangS

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The etiology in type 1 is thought to be a combination of lack of insulin (+/- glucagon and somatostatin), autoimmunity, autonomic neuropathy, and microvascular damage while the latter two contribute to pancreatic exocrine insufficiency in type 2 diabetes it has been suggested that this may explain why pancreatic exocrine insufficiency is more common in patients with type 1 diabetes (109)

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The density of CGRP-immunoreactive fibres in the TNC was increased in the CM and exendin(939) treated (CM + Exe(939)) groups compared with the sham group (Fig