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Nonetheless, this treatment still needs additional research, as the active metabolites causing levels of MMPs to drop were not identified
The beneficial effects of EPO in patients with toxic damage to the optic nerve secondary to methanol exposure are particularly presumed to be due to its ability to inhibit axon and RGC apoptosis, as well as the anti-oxidant activity exerted by an increase in the activity of two key enzymes in the anti-oxidant defense systemGSH and SOD (Pakravan et al
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Bioprocess Biosyst Eng 33:675682 Liao X, Deng T, Zhu Y, Du G, Chen J (2007) Enhancement of glutathione production by altering adenosine metabolism of Escherichia coli in a coupled ATP regeneration system with Saccharomyces cerevisiae
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doi:10.2174/138920211798120763 [4] Carta MG, Loviselli A, Hardoy MC, et al
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