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Our study demonstrates that DRP1 hyperactivation induces excessive mitochondrial fission, thereby synergistically enhancing the susceptibility of ATC cells to ferroptosis

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Dex, in this context, exerts neuroprotective effects against cerebral ischemic injury by interfering with mdm2-induced ubiquitination and degradation of PSD-95 through the modulation of HDAC5 and NPAS4 [70]

doi: 10.1016/j.neuint.2012.12.016 [DOI] [PubMed] [Google Scholar] 112.Stojkovska I, Wagner BM, Morrison BE

These findings position mTORC1 upstream of ISR signaling in this context