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These findings suggest that oxidative stress is intimately linked to a variety of pathophysiological processes, such as inflammation, oligodendrocyte abnormalities, mitochondrial dysfunction, hypoactive N -methyl-D-aspartate receptors and the impairment of fast-spiking gamma-aminobutyric acid interneurons.[bkyb1] Such self-sustaining mechanisms may progressively worsen producing the functional and structural consequences associated with schizophrenia

(2018) 34:e3027

National Toxicology Program

4a, b, e and f)

Therapeutic strategies targeting the ferroptosis pathway and metabolic vulnerabilities associated with ferroptosis have been reported in breast cancer and other types of cancer [13,25]

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