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The study also revealed that AS-IV could alleviate iron-dependent cell death in cardiomyocytes by activating the Nrf2 signaling pathway and enhancing the expression of GPx4, thereby attenuating oxidative stress induced by DOX (Luo et al., 2021a)

And its all because of a peptide thats made from the insulin cell by whats called a crinosome

Four other studies reported no statistically significant differences in glutathione levels in the posterior medial frontal cortex [23], mPFC [24] or anterior cingulate cortex (ACC) [25, 26]

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Notably, the ROS generated during ferroptosis further exacerbate mitochondrial damage (Jiang et al., 2021), forming a positive feedback loop of mitochondrial dysfunction-ferroptosis-excessive ROS-mitochondrial damage