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Once administered, the growth factors within the PRP stimulate stem cells, prompting the production of new, robust tissue

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Post-translational protein deimination signatures in serum and serum-extracellular vesicles of bos Taurus reveal immune, anti-pathogenic, anti-viral, metabolic and cancer-related pathways for deimination

The peptide's origin from gastric protective compounds may contribute to its robustness

Based on preclinical (animal and cellular) data only, inhibition of NNMT by 5-Amino-1MQ is theorised to: Raise intracellular NAD+ levels Activate SIRT1 and other sirtuins (proteins linked to metabolic regulation) Reduce fat cell (adipocyte) size and proliferation Improve insulin sensitivity These effects have been demonstrated in mouse models (Neelakantan et al., 2019), but robust human clinical trial data are currently absent