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Studies investigating ischemic kidney injury have shown that deletion of the alternative complement pathway attenuates injury from ischemia-reperfusion (97), as well as in C3a knockout mice, reduces the formation of NETs after ischemia, thereby protecting the kidney (96)

Despite their different physiological effects, all forms of I 2 have the potential to induce or exacerbate autoantibody formation

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Due to the lack of specific markers used in diagnostic and therapeutic evaluation, treatment for penile fibrotic diseases is still lacking