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It converts to S-adenosylmethionine (SAMe), which donates methyl groups to phosphatidylethanolamine, converting it to phosphatidylcholine

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All participants had a BMI 27 kg/m, established cardiovascular disease (prior MI, stroke, or peripheral artery disease), but no diabetes, making this the largest trial of a GLP-1RA in a non-diabetic, high-risk cardiovascular cohort

This, in turn, activates STAT3 signaling, leading to the upregulation of glycolysis-associated genes, notably independent of CHAC1s established role in ferroptosis induction (Pan et al., 2024)

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In the retatrutide trial data above, dysesthesia jumped from 8.8% at the 9 mg dose to 20.9% at the 12 mg dosea pattern that mirrors whats been seen with another GLP-1 medication at a higher dose