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NRF2 transcriptionally regulates key components of iron metabolism, including ferritin heavy and light chains (FTH1/FTL), which mediate iron sequestration, and ferroportin (SLC40A1), the primary cellular iron exporter [113]

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Therapy should therefore not be discontinued in patients with established CVD who do not achieve the weight-loss targets observed in clinical trials

Tirzepatide usually provides stronger metabolic benefits

GLP-1R also functions as a pleiotropic GPCR that can activate distinct signaling cascades depending on ligand bias and the local cellular environment [511]