In IBD, dysregulated redox signaling and metabolic dysfunction synergistically drive disease progression
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Abbreviations SJS: Stevens-Johnson Syndrome TEN: Toxic Epidermal Necrolysis FT1DM: Fulminant Type 1 Diabetes Mellitus IVIG: Intravenous immunoglobulin SCARs: Severe cutaneous adverse reactions DRESS: Drug reaction with eosinophilia and systemic symptoms AGEP: Acute generalized exanthematous pustulosis References Bastuji-Garin S, Rzany B, Stern RS, et al
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Figure 4 Alterations in the intracellular redox status are often associated with GSH depletion (Polonikov, 2020) and endogenous GSH deficiency, due either to decreased biosynthesis and/or increased consumption, is a marked contributor to the pathogenesis of numerous diseases via mechanisms including oxidative stress and inflammation
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