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The deletion of the gpoA gene resulted in a marked decrease in bacterial survival across various host cell types characterized by differing levels of H 2 O 2 production, including pneumocytes, macrophages, and neutrophils

We also found that the expression levels of related genes were also upregulated

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These results indicate that downregulation of the PPARa pathway reduces FABP1 expression, leading to alterations in GPX4 and ACSL4 levels that contribute to HMC ferroptosis and IgAN pathogenesis [161]

Interact J Med Res

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