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The underlying theory is straightforward: toxins, infections, oxidative stress, and chronic inflammation can damage cell membranes over time

Macrophage-derived CCL5 facilitates immune escape of colorectal cancer cells via the p65/STAT3-CSN5-PD-L1 pathway

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doi: 10.6065/apem.2244186.093 77 TaskaKGietka-CzernelMGlinickiPKozakowskiJ

10.1039/d1nr06342a 123 SelmiM.SalekA.BarbouraM.NjimL.TrabelsiA.LahmarA.et al (2023)

It reveals that altered glutathione and AA homeostasis induced by Trkb deletion in iSPNs is directly linked to early dopaminergic dysfunction and disrupted energy metabolism followed by age-dependent motor impairment and strongly supports the relevance of our model to elucidate the mechanisms underlying the vulnerability of iSPNs in neurodegenerative conditions