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Effects of Zinc Deficiency on Methylation and Glutathione Pathways Zinc deficiency can lead to a decrease in ceruloplasmin activity, leading to a decrease in copper levels

Cysteine, synthesized from homocysteine through the transsulfuration process, controls the production of GSH, the most prevalent antioxidant in mammals

As expected, the methylation level of UBE2m significantly decreased after PRMT1 knockdown and significantly increased after PRMT1 overexpression (Fig

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S.MilutinoviM.BrankoviS.Mihajilov-KrstevT.RandjeloviM.MiladinoviB.et al (2024)