Ferroptosis as a therapeutic target in CVD Targeting the core pathological drivers of ferroptosisincluding dysregulated iron metabolism, uncontrolled lipid peroxidation, impairment of the GPX4 antioxidant axis, and additional regulatory pathways such as FSP1CoQ 10 , GCH1BH4, and DHODHrepresents a promising strategy to suppress ferroptosis in cardiovascular disease
It is not an approved therapy in most regions
Janoshik COA verified
Dopamine and serotonin system interactions
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