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According to a previous report, fluoroquinolones generated ROS in human tenocytes through mitochondrial membrane damage, which was mitigated by MitoQ, a mitochondria-targeted antioxidant (Lowes et al., 2009)

10.1001/jama.285.4.413 6

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We modeled donor-specific effects by treating each donor as an independent batch in the design formula (~batch + treatment) during regression analysis

The one that is most easy to obtain is Sermorelin

468 used a conjugate of the cytoplasmically localized internalized peptide (CLIP6) and PNA to treat GBM cells, considerably upregulating the tumor suppressor Mnk2a and promoting cancer cell death