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This auto-oxidation process generates toxic reactive oxygen species (ROS), quinones, and free radicals that damage intracellular organelles, deplete cellular glutathione, and ultimately trigger apoptotic cell death (Lohr et al., 2014

Glucagon-like peptide-1 inhibits vascular smooth muscle cell dedifferentiation through mitochondrial dynamics regulation

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Preclinical studies in rodents show it enhances synaptic connectivity and spatial memory retention with reported potency millions of times greater than BDNF

10.1002/cbin.10543 Summary Keywords berberine, berberine metabolite, intestinal-hepatic-vascular tissue, pharmacological mechanism, lipid metabolic disease Citation Cai Y, Yang Q, Yu Y, Yang F, Bai R and Fan X (2023) Efficacy and underlying mechanisms of berberine against lipid metabolic diseases: a review

ACSL3) and in the mode of delivery (paracrine induction vs