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Many molecular targets and signaling pathways, such as nuclear factor kappa-B (NF-B), phosphatidylinositol 3 kinase/protein kinase B (PI3K/AKT), Wnt/-catenin, adenosine 5-monophosphate-activated protein kinase (AMPK), toll-like receptor (TLR), Janus kinase/signal transducer and activator of transcription (JAK/STAT), long noncoding RNAs (lncRNAs), and microRNAs (miRNAs), play crucial roles in the progression of UC

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(27) Specifically, dulaglutide, exenatide, liraglutide, semaglutide, and lixisenatide were compared

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