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In contrast, liposomal supplements leverage lipid bilayer vesicles that encapsulate and protect active ingredients as they travel through the digestive tract

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Using C2C12 mouse myoblasts, the study confirmed that the MGF E-domain peptide increases myoblast proliferation while inhibiting terminal differentiation and critically, that this effect was maintained in the presence of a neutralising anti-IGF-1R antibody directly establishing that MGF E-peptide signals via an IGF-1R-independent mechanism

Available from: Reiss AB, Gulkarov S, Lau R, Klek SP, Srivastava A, Renna HA, De Leon J