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ROS also promotes osteoclast differentiation through multiple pathways, including MAPK, IP3, and NFkB [18], leading to osteoclast maturation via activation of genes like NFATC1 [19]

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Furthermore, in vitro studies have demonstrated that high-glucose conditions dose-dependently suppress PPAR activity in astrocytes, increasing their susceptibility to ferroptosis, and gemfibrozil, a PPAR agonist, inhibits ferroptosis in db/db mice and astrocytes [160]