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Its upregulation may therefore serve as an intrinsic feedback mechanism to prevent exhaustion or toxicity caused by overactivation

The current status of homocysteine as a risk factor for cardiovascular disease: A mini review

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Turner RS, Thomas RG, Craft S, et al

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Using only the differentially expressed genes with LPS treatment, to determine genes rescued with NR treatment, a subsequent analysis using DESeq2-identified genes that changed between LPS and LPS with NR groups with an FDR of 10% was performed