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However, Randles proposal is still physiologically relevant for the control of oxidative substrate selection and can be seen as a cell-autonomous response to the overall regulation of substrate availability, promoting efficient glucose oxidation in the fed state and fatty acid oxidation in the fasted state [35]

If IGF-1 remains unchanged, it suggests poor timing, inadequate dosing, or receptor resistance

Future work should integrate advanced models with complementary high-performance tools to overcome current limitations and unlock their therapeutic promise

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In myoblast culture models, researchers observed that IGF-1 LR3 is associated with increased myotube formation, protein synthesis rates, and satellite cell activation markers

M.BastianelliS.GiustariniD.PierucciS.BustiC.et al