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Mechanistically, this combination therapy modulates key mediators such as thromboxane A2 (TXA2) and prostacyclin (PGI2), including their downstream effector cyclooxygenase-2 (COX-2), thereby contributing to its antithrombotic properties

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166,190 Further, in gastrointestinal cancer cells, genetic inhibition or pharmacological treatment of CPT2 with perhexiline disrupts NADPH and promotes cell apoptosis after oxaliplatin treatment

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This suppression subsequently leads to a reduction in intestinal Th17 immune cells

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