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In gastric cancer, catalpol inhibits the proliferation and migration of human gastric cancer cell HGC-27 induced by TGF-1 by targeting the MAPK/ERK signaling pathway

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Through diverse molecular mechanisms, it interferes with normal cellular processes such as transformation, senescence, programmed cell death, and proliferation, thereby directly contributing to the development of various human diseases (Table 3)

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Treatment of Peyronies disease with collagenase clostridium histolyticum and vacuum therapy: a randomized, Open-label pilot study

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Regulation of FoxO activity by CBP/p300-mediated acetylation

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Typical daily range: 2050 mcg once daily subcutaneously, raised gradually over an 812 week course

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