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Notably, CUL5 exhibits dual regulatory functions in various cancers, and its expression level correlates differently with prognosis depending on tumor type
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However, the gshF copy number increase didnt further improve GSH concentration in W303-1b/F, indicating that a reasonable rather than high copy number of gshF gene driven by GAP promoter is sufficient for the highest expression levels and synthetic capacity of GSH through one pathway is limited
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