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SPAG5-AS1 inhibited autophagy and aggravated apoptosis of podocytes via SPAG5/AKT/mTOR pathway

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The peptide appeared to promote tendon fibroblast proliferation and accelerate the transition through the organisational phase of healing

Pre-clinical rodent models of tendon rupture have shown combined BPC-157 and TB-500 protocols to produce faster return of tensile strength and superior histological organisation compared to either single-peptide treatment or untreated controls