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Adding the second compound too early often leads to intense nausea and a forced step-down on one or both compounds

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Then we turn to a discussion of clinical and pre-clinical findings of redox impairment that induce brain pathology in schizophrenia, through mechanisms that likely involve aberrant inflammatory responses, mitochondrial dysfunction, oligodendrocyte abnormalities, epigenetic changes, hypoactive N -methyl-D-aspartate (NMDA) glutamate receptors and the impairment of fast-spiking gamma-aminobutyric acid (GABA) interneurons ( see Figure 1)

512,513 Correcting exon 7 splicing in SMN2 can compensate for the lack of SMN1, producing functional SMN protein and improving symptoms