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As illustrated in Figure 1 , ferroptosis-derived signals such as lipid peroxides (e.g., MDA and 4-HNE) and DAMPs (e.g., HMGB1, IL-33) activate immune cells, including M1 macrophages and Th17 cells
2009), which caused complete degradation of the substance (Patel et al
Indeed, superoxide production has previously been demonstrated to be linked to the susceptibility of MNA NB cells to DPI, resulting in cytochrome c release, caspase activation, cytochrome c release and cell death ( 2 O 2
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