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As discussed previously, NO synthesis is likely increased during at least some stages of chronic sleep restriction, pointing to NO as one possible mediator linking sleep disturbance to neurodegenerative pathology

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Wound healing: The ability of Pal-GHK to reduce inflammation, promote angiogenesis, and stimulate the regeneration of tissues enables it to accelerate the rate at which wounds heal [7,8]

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Loss of aqp4 polarized localization with loss of beta-dystroglycan immunoreactivity may induce brain edema following intracerebral hemorrhage