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These findings suggest that GLP-1RAs, like NLY01, can lessen AD pathology by targeting neuroinflammation related to microglial activation and reactive astrocytes, thus restoring memory functions and offering neuroprotective benefits [50]
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Following brain injury, CD4 T cells differentiate into various effector subsets, each of which secretes distinct cytokines that exert either proinflammatory or anti-inflammatory effects [157, 158]
