The presence of GLP-1 receptors in the brain has prompted considerable scientific interest in understanding how this hormone influences neurological function
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Chronic peripheral hyperinsulinemia is expected to cause the downregulation of insulin receptors at the NVU and reduction in the insulin entry into brain, as reflected in lower brain insulin concentration in AD patients [214,215]
Clin Chim Acta 1990;187:329-32
found that endothelial cell dysfunction, which is characterized by low nitric oxide (NO) bioavailability, greater reactive oxygen species production, the activation of abscission enzymes, and impaired extracellular lysosome-associated organelle function, triggers the degradation of the EG (57), leading to shear stress defects
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