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Depletion of GSH in glial cells induces neurotoxicity: relevance to aging and degenerative neurological diseases
LoguercioCPiscopoPGuerrieroCGirolamoVDDisalvoDdel Vecchio BlancoC
Furthermore, GSTP1-1 has also been shown to bind and inhibit tumor necrosis factor receptor-associated factor 2 (TRAF2), an upstream activator of JNK, thus blocking the MAPK/JNK signaling cascade at multiple levels 29
We use the model to simulate the metabolic profiles previously observed in Down syndrome and autism and compare the model results to clinical data
Alterations in GSSG generation, recycling, and/or efflux have all been reported to be associated with correspondingly altered cell vulnerability to cell death in response to various cellular stresses
Efficient delivery of Temozolomide using ultrasmall large-pore silica nanoparticles for glioblastoma
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