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Pharmacokinetic (PK) profiling in both species revealed linear doseexposure relationships, with [H] BPC157 rapidly metabolized into small peptides and ultimately tritium-labeled proline for normal amino acid catabolism/excretion [11]

In parallel, VDAC1 oligomerization has been implicated in ferroptosis-associated mitochondrial dysfunction, and pharmacological blockade of VDAC1 oligomerization suppresses mitochondrial ROS accumulation and lipid peroxidation during ferroptosis [99]

Pause the supplement for a week and observe

Higher doses and lifestyle changes improve outcomes

The Lancet, 398(10295), 143155