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Studies indicate that the glycocalyx is critical in treating sepsis, malaria, traumatic brain injury and coronary atherosclerosis [10,11,12,13]

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Furthermore, semaglutide prevents MASLD/MASH-driven HCC progression in metabolic preclinical models, partly by correcting adipokine imbalances (low adiponectin, high leptin) associated with insulin resistance, which removes a key chronic inflammatory and pro-oncogenic signal linked to the disease (Kojima et al., 2020)

Some studies have noted that although NK cells can clear certain damaged chondrocytes or pathogens in early OA, they also release ROS under highly inflammatory conditions, aggravating oxidative stress and indirectly activating ferroptosis pathways (116)