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To solve or reduce these problems, cells use repair and detoxification pathways as well as surveillance mechanisms, called checkpoints, which serve to detect the problem and coordinate repair with chromosome segregation and progression through the cell cycle (see Figure 3

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Gut-associated lymphoid tissue (GALT) and local immune cells can detect external stimuli and release inflammatory mediators such as interleukin-6 (IL-6) and tumor necrosis factor- (TNF-)

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