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doi: 10.1579/0044-7447(2007)36[94:SGAH]2.0.CO;2

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We identify mitochondrial trifunctional protein- (MTP) as a binding partner of GLP-1(2836) and demonstrate that the ability of GLP-1(2836) to shift substrate utilization from oxygen-consuming fatty acid metabolism toward oxygen-sparing glycolysis and glucose oxidation and to increase cAMP levels is dependent on MTP

The outer mitochondrial membrane is relatively permeable, but the inner mitochondrial membrane is strictly impermeable to long-chain fatty acids