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It modulates gene expression broadly downregulating NF-B inflammatory programs while upregulating antioxidant and repair pathways

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Others directly inhibit pro-inflammatory signaling either by blocking cytokine release or by binding receptors that shift immune cells into a less reactive state

Similarly, Selank is applied in investigations of anxiolytic signaling and immune-neuroendocrine interactions, often within GABAergic and cytokine-related pathways

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