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Altogether, these elastase-mediated processes lead to the development of an emphysema-like phenotype

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IL-1 then activates pyroptosis by binding to its receptor on the cell membrane, resulting in the formation of gasdermin D (GSDMD) pores that cause cell membrane rupture and the release of proinflammatory cytokines and damage-associated molecular patterns (DAMPs), such as high-mobility group box 1, heat shock proteins, and fibronectin advanced glycation end-products IL-33

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On the antioxidant effects of ascorbic acid and glutathione

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