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In addition, high-salt diets have led to histologically detectable intestinal inflammation in mice models, as confirmed by goblet cell depletion, ulceration, erosion, and destruction of the mucosal layer (Aguiar et al

The presence of specific bacterial taxa in human feces was associated with both plasma TMAO concentration and dietary status

This causes further hyperinsulinemia, hypertriglyceridemia, increased IR in these tissues [27], and risk of T2DM [28]

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Increased severity of inflammation correlates with elevated expression of TRPV1 nerve fibers and nerve growth factor on interstitial cystitis/bladder pain syndrome

Thanks to all authors for their contributions to this study