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[25] In addition, in TBI and hypoxia models, NAC has repressed the development of secondary injury through the modulation of important molecular pathways like the nuclear factor erythroid 2related factor 2 pathway and the upregulation of GPX4, an important enzyme that suppresses ferroptosis

31 Drug-induced vasculitis typically presents 1 to 3 weeks after drug initiation and is usually self-limited

Thus, there exists a tight coupling between the glutamatergic system and brain energy metabolism via astrocytic functions

A study published in Neurology by Stanley et al

CAB processed and prepared the data

detected no variation in the levels of F2-isoprostane or in the use of antidepressants between the control group and individuals with depression