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Mouse islets cultured in 1% O 2 activated pro-apoptotic ER stress pathways, and quantification of autophagosomes and autolysosomes in CAG-RFP-EGFP-LC3 islet cells revealed that hypoxia significantly disrupted autophagic flux

10.1080/15622975.2017.1308552 174 MurakamiK.IrieK.MorimotoA.OhigashiH.ShindoM.NagaoM.et al (2002)

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