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Reshma Ramachandran, a Yale medical school professor and the new study's senior author, told the Washington Post

Initially, it disrupts chromosomal cohesion by downregulating PP2A and impairing Rec8 dephosphorylation

There appears to be an anomalous storage of mitochondrial ATP synthase subunit c that is neither the result of mutation nor enhanced expression of the protein but rather a slower degradation of the mitochondrial ATP synthase in comparison with normal cells [116]

This is partly because excess thiamine is quickly excreted from the body in urine

doi: 10.4088/JCP.13m08454