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[PMID:26590474] Usui N, Kamiyama M, Tani G, et al

To further confirm that AKT inactivation increased MEK1/2 phosphorylation, we knocked down AKT1 and AKT2 expression by transfection of akt1 and akt2 siRNAs

Such phenotypic heterogeneityshaped by both intrinsic programs and extrinsic cuesunderscores the limitations of traditional binary models and highlights the need for precise classification systems

From endothelial dysfunction to arterial stiffness in diabetes mellitus

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