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Keywords: lipoic acid, acetyl-L-carnitine, mitochondrial dysfunction, oxidative damage, Parkinsons disease Introduction Increasing evidence indicates that mitochondrial dysfunction and oxidative stress play vital roles in the pathogenesis of sporadic and familial forms of Parkinsons disease (PD) [1, 2]

They may develop a loss of energy and appetite and failure to thrive, which if not treated on time can lead to coma and death

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Short-term Western diet intake promotes IL-23mediated skin and joint inflammation accompanied by changes to the gut microbiota in mice