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In both animal models and ALS patients, the endogenous T lymphocytes, specifically Treg cells, increased at an early slowly progressing disease stage, and numbers decreased in conjunction with rapid ALS progression, likely due to the loss of FoxP3, a transcription factor for Treg function [54, 58]

And NLRP3 inhibitors can significantly improve proteinuria and renal function in mice, underscoring the importance of the NLRP3 inflammasome in kidney disease [183, 184]

PMCID: PMC8540239

DOI: 10.1007/s10517-014-2544-7 15